The glomerular filtration rate remains relatively constant.
肾小球滤过率也能保持恒定.
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Glomerular changes are both frequent and severe.
肾小体的病变是常见而严重的.
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The development of severs hypertention in glomerular disease is an ominous s ign.
肾小球疾病时发生严重高血压是一种不良的现象.
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Cast preparation of the glomerular vessels ( glomerulus ) in a renal corpuscle.
铸造编制肾小球船只 ( 肾小球 ) 在肾小体.
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Results Among 42 cases electrophoresis indicated 27 glomerular proteinuria; 15 mixed proteinuria.
结果42例中27例为肾小球性蛋白尿, 15例为混合性蛋白尿.
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Magnified portion of a section showing the glomerular capillaries and Bowman's space.
放大部分的一节显示肾小球毛细血管和鲍曼的空间.
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The branches reach to glomerular basal membrane.
分支机构达到肾小球基底膜.
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DNA fragmentation localized to glomerular cells.
定位于肾小球细胞的DNA断裂.
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Results consistently demonstrated that DNA colocalized with autoantibodies in glomerular membrane associated EDS.
结果始终表明,DNA与肾小球膜电子致密带的自身抗体的共区域.
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Results In normal kidney tissues, ILK was mainly located in the glomerular visceral epithelialcells.
结果在正常组肾组织, ILK主要表达于肾小球脏层上皮细胞.
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Immune complexes nephritis is a typical nephritis model, its pathological change finally lead to glomerular sclerosis.
免疫复合物型肾炎是一种经典的肾炎模型, 其病理改变最终将导致肾小球硬化.
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Objective : To investigate the mechanism inducing apptosis of human glomerular mesangial cells ( Mcs ) by anti Fas antibody.
目的: 研究抗Fas抗体 对人肾小球系膜细胞的致凋亡作用,探讨Fas? FasL 在肾脏损伤中的作用.
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Methods Changes relating to glomerular function, structure and glomerular basement membrane ultrastructure were observed.
方法观察低分子肝素对肾小球功能 、 形态及肾小球基底膜超微结构的影响.
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Results In normal kidney tissues, ILK was mainly located in the glomerular visceral epithelial cells.
结果在正常组肾组织, ilk主要表达于肾小球脏层上皮细胞.
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Furthermore, glomerular damaged showed a significant correlation with tubular DNA damage in proliferatie GN.
另外, 在增生性肾小球肾炎中损伤的肾小球和肾小管DNA断裂显著相关.
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